Title: Alternatively activated macrophages in infection and autoimmunity.
Summary: "Women may be at an increased risk of M2-mediated infection and autoimmunity due to estrogen's ability to increase Th2 responses."
Citation: Fairweather, D. and Cihakova, D. (2009). Alternatively activated macrophages in infection and autoimmunity. Journal of autoimmunity. http://www.citeulike.org/user/HEIRS/article/5948117
Showing posts with label pathogenic infections. Show all posts
Showing posts with label pathogenic infections. Show all posts
Friday, October 16, 2009
Fats Interact With Immune Complex to Activate Inflammation -May Aggravate Autoimmune and Pathogen Infection
HEIRS Environmental Illness Research Blog: Infection/Endotoxin in Chronic Fatigue, Fibromyalgia and Multiple Chemical Sensitivity.
The interaction between toll-like receptors and fatty acids "engage" the immune complex Il-1b and its receptor to activate other inflammatory processes in the pancreas.
Böni-Schnetzler, M., Boller, S., Debray, S., Bouzakri, K., Meier, D. T., Prazak, R., Kerr-Conte, J., Pattou, F., Ehses, J. A., Schuit, F. C., and Donath, M. Y. (2009). Free fatty acids induce a proinflammatory response in islets via the abundantly expressed interleukin-1 receptor i. Endocrinology, pages en.2009-0543+. http://www.citeulike.org/user/HEIRS/article/5922922
The interaction between toll-like receptors and fatty acids "engage" the immune complex Il-1b and its receptor to activate other inflammatory processes in the pancreas.
Böni-Schnetzler, M., Boller, S., Debray, S., Bouzakri, K., Meier, D. T., Prazak, R., Kerr-Conte, J., Pattou, F., Ehses, J. A., Schuit, F. C., and Donath, M. Y. (2009). Free fatty acids induce a proinflammatory response in islets via the abundantly expressed interleukin-1 receptor i. Endocrinology, pages en.2009-0543+. http://www.citeulike.org/user/HEIRS/article/5922922
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