Summary: "enhanced MAO-A activity coupled with increased intramyocardial norepinephrine availability results in increased reactive oxygen species generation which can lead to cardiac dysfunction from chronic stress."
Increased Norepinephrine Breakdown Adverse for Heart. Mpdern Medicine. Retrieved January 14, 2009.
Thimmulappa, R. K., Mai, K. H., Srisuma, S., Kensler, T. W., Yamamoto, M., and Biswal, S. (2002). Identification of nrf2-regulated genes induced by the chemopreventive agent sulforaphane by oligonucleotide microarray. Cancer Res, 62(18):5196-5203.
http://www.citeulike.org/user/HEIRS/article/3728059
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