"These findings suggest that the EAAC1−/− mouse may be a useful model of the chronic neuronal oxidative stress that occurs in PD. The salutary effects of N-acetylcysteine in this mouse model provide an impetus for clinical evaluation of glutathione repletion in PD. ANN NEUROL 2010"
Read more: CiteULike: N-acetylcysteine prevents loss of dopaminergic neurons in the EAAC1−/− mouse:
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