Summary: This nuclear translocation of NF-kappaB may promote viral latency by negatively regulating BZLF1 transcriptional activity. In situations where p65 activity is limiting in comparison to BZLF1, the ability of BZLF1 to inhibit p65 transcriptional function may protect the virus from the host immune system during the lytic form of infection.
Morrison, T. E. and Kenney, S. C. (2004). Bzlf1, an epstein-barr virus immediate-early protein, induces p65 nuclear translocation while inhibiting p65 transcriptional function. Virology, 328(2):219-232. http://www.citeulike.org/user/HEIRS/article/6463038
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